In T2DM, elevated LC3-II and p62 levels suggest that autophagic flux is active but may be dysfunctional due to inefficient degradation of the autophagosomes
Took away some of my hope to reverse this disease!
GLP-1 receptor agonists reduce TNF-alpha production in multiple tissue types, including immune cells, cardiac tissue, and brain tissue
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Inhibitors of histone deacetylases induce tumor-selective apoptosis through activation of the death receptor pathway
