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The mechanisms by which inflammation affects tight junction structure and function and transcellular mechanisms such as endocytosis are largely unknown, although reproducible correlations have been observed between the severity of experimental and clinical disease and on the one hand and transepithelial electrical resistance, transport of paracellular markers, and systemic LPS concentrations and on the other [42, 86]

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Together, these observations establish that perioperative mitochondrial dysfunction arises from the integrated action of ROS, RNS, Ca 2+ overload, and mPTP dynamicsnot ROS alone

In experimental models, glutathione is used to study oxidative stress, detoxification, immune function, neurobiology, cartilage and joint health, ocular physiology, and skin-aging mechanisms