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Pathways associated with muscle development and extracellular matrix remodeling were significantly upregulated, potentially reflecting compensatory tissue repair mechanisms in response to neuromuscular inhibition (Supplementary Fig

The non-negotiable: GLP-1 without strength training is a bad idea. How to protect muscle with nutrition and exercise is reviewed

Epithelial cells, ECs, and vascular smooth muscle cells (VSMCs) in multiple organ systems may be the primary targets of TMAO-induced autophagy-dependent cell death

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