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With the development of genetic testing technologies such as genome-wide association studies (GWAS) and next-generation sequencing (NGS), it has been discovered that approximately 127 locus abnormalities in the human genome are associated with the onset and progression of obesity [4] , as along with more than 550 monogenic mutations related to obesity [5]

A major regulator of the rate of NADPH production by the PPP in response to oxidative stress is the allosteric activation of G6PDH

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