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Mechanism of action: 1) activation of nitric oxide (NO) system improves circulation in damaged tissues (Sikiric et al., J Physiol Paris, 1999)

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7 This is in part caused by its counteracting effect on DPP-4, a GLP-1 inhibitor, prolonging the GLP-1R signal and maintaining insulin secretion

[Nitric oxide synthase activity and nitrates/nitrites level in brain regions during the spontaneous morphine withdrawal in rats]

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described a strong correlation between the antioxidant activity of polyphenols and their affinity for Fe(II) ions, suggesting that the suppression of the Fenton reaction is probably due to ion chelation [93]