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The results indicated that the proportion of mature DCs (CD80 + CD86 + ) in the TDLNs of mice treated with NP4 (82.8%) was approximately 1.46 times higher than that in the mice treated with NP2 (56.7%) (Fig

Polymeric nanoparticles in gene therapy: new avenues of design and optimization for delivery applications

Alogliptin Attenuates Lipopolysaccharide-Induced Neuroinflammation in Mice Through Modulation of TLR4/MYD88/NF-kappaB and miRNA-155/SOCS-1 Signaling Pathways

If these properties are conserved, we speculate that in humans, as inflammaging favours fibrosis, fibrosis may in turn promote Notch hyperactivation

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