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In this study, we have investigated the capacity of L-cells to interpret bacterial signals from the gut lumen and activate host colonic afferents and intrinsic neurons by secreting GLP-1

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Subsequently, transmission electron microscope (TEM) observations showed that LPS treatment led to mitochondrial shrinkage, increased membrane density and reduced volume, and 2,3-cGAMP further aggravated this pathological change, while C-176 or GLP-1RAs could effectively alleviate the above-mentioned mitochondrial damage (Fig

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Molecular regulation of autophagy machinery by mTOR-dependent and -independent pathways