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For example, microsatellite stable colorectal cancer (MSS CRC) cells accumulate distal cholesterol precursors (DCPs) due to asynchronous cholesterol biosynthesis, polarizing T cells toward a pro-tumorigenic Th17 phenotype and promoting immune suppression

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Currently, there are limited therapeutic options for the disease because of limited understanding of its molecular mechanism

demonstrated increased GSH levels as well as decreased nuclear factor kappa B activity corresponding with overall decreased oxidative stress on the cardiomyocytes and decreased myocyte apoptosis [47]