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The dual mechanism produces additive or combined effects on energy balance -- GIPR activation in the CNS may enhance the anorectic signals from GLP-1R activation, and peripheral GIPR signaling improves adipose tissue function and lipid handling in ways GLP-1 alone does not
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The findings may pave the way toward the development of 15LO1 inhibitors as asthma treatments
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This evidence indicated that in fish, increasing the catabolism of lipids decreases the energy portion sourced from glucose degradation, resulting in increased gluconeogenesis and glycogen synthesis
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