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Accordingly, CB2R serves an adaptive function, and its upregulation represents a marker of microglial and astrocytic activation during neuroinflammatory processes (Vuic et al., 2022)

As a consequence of the competition between these and many other GSH-consuming pathways, on one hand, the raging inflammation and oxidative stress triggered by the viral infection steals GSH from core functions such as NO-dependent vasodilatation

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For patients considering their options: if oxidative stress is clinically evident (ask your physician about inflammatory markers like hsCRP, liver enzymes, or oxidised LDL), glutathione or NAC supplementation is reasonable adjunctive support

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