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It contributes to energy production, anaplerosis, and biosynthesis of amino acids and nucleotides

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( d ) Left: cell count as a percentage of the total per cluster per genotype, right: cell count per slice per cluster per genotype (GnasWT: n = 1,129 cells, 5 slices, 3 mice, 2 males

Manage complications on a case-by-case basis, e.g.: Complications [1] [2] Inflammatory Small bowel diverticulitis (most common complication of jejunoileal diverticulosis) [1] Peritonitis (due to gastrointestinal perforation) Abscess formation Hemorrhage (most common complication of duodenal diverticulosis) [1] Mechanical bowel obstruction due to, e.g.: Enterolith formation and impaction Volvulus Intussusception Stricture or adhesions from prior inflammation Compression (e.g., resulting from a distended diverticulum) Nonmechanical bowel obstruction (e.g., from intestinal dyskinesia) Small intestinal bacterial overgrowth leading to, e.g.: Malabsorption Nutritional deficiencies (e.g., vitamin B 12 deficiency and fat-soluble vitamin deficiencies ) Anemia Pancreaticobiliary disorders (associated with periampullary diverticula) Benign pneumoperitoneum Small bowel diverticulitis Definition [1] [2] The inflammation of one or more small bowel diverticula (most commonly jejunoileal diverticula) Caused by stasis of contents within the diverticulum, resulting in venous outflow obstruction and infection Epidemiology [3] Typically affects older adults Most common complication of jejunoileal diverticulosis Etiology [1] [2] [3] Obstruction of the diverticulum due to, e.g.: Predisposing factors include: Foreign body Cholelithiasis Diverticular ulceration Blunt trauma Clinical features [1] [2] [3] Presentation is often nonspecific and can mimic other causes of an acute abdomen