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Differential mitochondrial roles for alpha-synuclein in DRP1-dependent fission and PINK1/Parkin-mediated oxidation

Animal studies demonstrate that chronic tesofensine treatment produces sustained weight loss even as tolerance to the hypophagic effect gradually develops, indicating additional factors beyond appetite regulation contribute to maintaining maximal weight loss

Additionally, Ginsenoside Rg1 reduces mitochondrial oxidative stress by inhibiting TXNIP/NLRP3, downregulates mitophagy proteins such as PINK1/Parkin to maintain mitochondrial homeostasis, and simultaneously increases the activity of antioxidant enzymes and enhances the eNOS/NO pathway to improve endothelial function and pulmonary vascular remodeling (Ran et al., 2024

Subsequent work in lean PCOS extended these metabolic and androgenic improvements, although sample sizes were modest and follow-up was short