The elevated antioxidant capacity driven by persistent NRF2 activation and enhanced glutathione (GSH) and thioredoxin (TRX) systems enables cancer cells to withstand endogenous oxidative stress and promotes metastatic behavior
Novel insight to neuroprotective potential of curcumin: a mechanistic review of possible involvement of mitochondrial biogenesis and PI3/Akt/ GSK3 or PI3/Akt/CREB/BDNF signaling pathways
Insulin resistance as well as the hypertensive response can cause damage to the structural integrity and transport function of the bloodbrain barrier, increase the permeability of the bloodbrain barrier (24), and the entry of free fatty acids (FFA) and other plasma components into the brain tissue to disrupt the homeostatic balance of the brain, as well as the entry of immune cells and inflammatory factors in the peripheral circulation into the CNS, which can cause further neuronal damage, thus leading to cognitive dysfunction (19)
subcutaneous), and other pathological factors
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