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HS is strongly associated with immune abnormalities, oxidative damage, 8 and elevated cytokine pathways, including tumor necrosis factor-alpha (TNF-), interleukin-17A (IL-17A), interleukin-17F (IL-17F), interleukin-1 beta (IL-1), interleukin-23 (IL-23), and interleukin-26 (IL-26)

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Consequently, several studies have indicated a correlation of IGF-1R expression with disease development [70,71]

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