[DOI] [PMC free article] [PubMed] [Google Scholar] 21.Mynster Kronborg T, Frohnert Hansen J, Nielsen CH, et al
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Cells were washed, colleted with 1 mM EDTA in PBS, pelleted (500 g for 5 min) and re-suspended in isolation medium (320 mM sucrose, 10 mM TrisHCl, 1 mM EDTA, pH 7.4)
doi:10.1042/BJ20081386, PMID: 19061483 Cox AG, Winterbourn CC, Hampton MB (2009) Mitochondrial peroxiredoxin involvement in antioxidant defence and redox signalling
In this context Hurthle cells do not represent a discrete adenoma
The anti-endometriosis effects involve plentiful molecular mechanisms and targets, including anti-proliferative (CCNE1, CDK2, and CDK4), anti-inflammatory (PGE2, COX-2, TNF-, NO, IL-1, IL-6, and IL-8), anti-angiogenic (VEGF, matrix metalloproteinase and Ang-1), pro-apoptotic (Bax, Bak, Bcl-2, Bcl-xL), autophagy (Beclin 1, LC3-II/LC3-I), endoplasmic reticulum stress, endoplasmic reticulum apoptotic pathway, mitochondrial apoptotic pathway and caspase cascade, anti-migratory invasion (MMP-2, MMP-9, TIMP1, E-cadherin, N-cadherin, Snail and Slug), regulation of estrogen and progesterone receptors, and anti-fibrotic (E-calmodulin, -SMA, type I collagen, wave proteins and fibronectin)
