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TENS also contributes to pain relief by inducing the release of endogenous opioids [87,90]

KPV, the C-terminal tripeptide of -MSH, is studied for damping pro-inflammatory signaling so remodeling can proceed on a calmer background

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In this review article, we focus on the signaling through which uremic toxins, particularly indoxyl sulfatean independent risk factor for cardiovascular complications in CKD patients, modulate macrophage activation and function, and how these changes contribute to vascular inflammation, leading to the increased cardiovascular risk