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This nanomedicine induces mitochondrial membrane potential collapse, ATP depletion, and excessive accumulation of ROS, ultimately triggering mitochondria-dependent apoptosis, thereby effectively reversing oxaliplatin resistance [146]

Additionally, a co-culture system of MCECs and MPMs revealed that GHK-Cu facilitated MCECs healing, impaired by DSS, by upregulating ZO-1 and Occludin expression

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