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Silencing TfR1 not only reduces chondrocytic iron content and oxidative stress but also promotes mitophagy and inhibits the mtDNA/cGAS/STING inflammatory axis, thereby breaking the cycle between ferroptosis and immune activation

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Associated with metabolic syndromes, NAFLD is increasingly being recognized as a major cause of liver disease

Their complementary mechanisms one acting primarily through growth factor upregulation and GI-cytoprotection, the other through actin polymerization and systemic cell migration make them uniquely interesting both individually and as a combined research subject