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On other hand, prooxidant therapies, including ionizing radiation and chemotherapeutic agents, are widely used in clinics, based on the rationale that a further oxidative stimulus added to the constitutive oxidative stress in tumor cells should, in fact, cause the collapse of the antioxidant systems, leading to cell death [6]

Thymosin 4: actin-sequestering protein moonlights to repair injured tissues

Its primary job is to neutralize free radicals

Dual targeting of glutaminase 1 and thymidylate synthase elicits death synergistically in NSCLC

Beyond 8 weeks, diminishing returns set in and risk of tolerance increases

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