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For instance, Laurent et al., 2004 [65] showed that NAC at 150 mg/kg daily for 2 months could reduce lipid oxidation and increase the intracellular and the mitochondrial pool of GSH
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In laboratory and translational models, cagrilintide is therefore used to explore how peptide-mediated signaling affects glucose metabolism, energy regulation, and cross-talk between appetite pathways and peripheral metabolic tissues [4][6]
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Physiopathological consequences with clinical expression have been observed in different systems, such as that recently found by Chang-Hoon et al., in which patients with fatty liver had lower lung function as measured by FVC [45]
