Ready to start your weight loss journey with medication that actually works
The result: Increased lipolysis : Fat cells release stored fatty acids for use as fuel Inhibited lipogenesis : Dietary fats are less readily converted to stored body fat No significant IGF-1 stimulation : Means no growth of existing tumors or cancer risk amplification that concerns some clinicians with full GH No significant glucose disruption : Unlike full HGH, which causes insulin resistance at therapeutic doses, AOD-9604 shows minimal glucose impact in studies This makes it uniquely well-suited for fat loss use cases where full HGH would be too blunt an instrument
This inhibition preserves cellular NAD+ levels, enhancing mitochondrial function, improving fat oxidation, and supporting overall metabolic health
H., Ho, K
References Nature Medicine (2014) Nicotinamide N-methyltransferase knockdown protects against diet-induced obesity PMC (2024) Nicotinamide N-methyltransferase inhibition mitigates obesity-related metabolic dysfunctions Frontiers in Pharmacology (2024) NNMT: a novel therapeutic target for metabolic syndrome Creative Peptides Peptide stability and storage guidelines for lyophilized compounds PubMed (2021) LC-MS/MS assay for 5-amino-1-methylquinolinium: pharmacokinetic and oral bioavailability study PMC Subcutaneous drug injection review: pharmacologic considerations NCBI Bookshelf Best practices for injection (asepsis, preparation, and administration) ResearchGate (2021) Combined NNMT inhibition and reduced-calorie diet normalizes body composition in obese mice PMC (2022) Reduced calorie diet combined with NNMT inhibition establishes a distinct microbiome in DIO mice Scientific Reports / NMN.com Role of NNMT inhibition in muscle strength: enhanced grip strength with exercise Swolverine 5-Amino-1MQ mechanism, benefits, stacking and cycling guide CDC Vaccine administration: subcutaneous route (angle/site
Fat storage versus fat burning NNMT does not just affect NAD+ levels
