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The role of the metabolic polymorphism of GSTM1 and GSTT1 in the susceptibility to diabetic nephropathy [Thesis]

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GSH is generated by de novo synthesis in a process mediated by two ATP-dependent ligases (glutamate-cysteine ligase and glutathione synthase), as well as by the regeneration of GSH from GSSG, a process catalyzed by glutathione disulfide reductase

G6PD deficiency is an X-linked recessive hereditary condition that impairs the ability of red blood cells to handle oxidative stress

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