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Rac1 and Stathmin but not EB1 are required for invasion of breast cancer cells in response to IGF-I

doi: 10.1007/s00203-023-03589-7 37 VickersNJ

Liew Z, et al

The minimum network analysis of the analyzed steady-state metabolites and their interaction indicated that potential changes are centered on NADH (betweenness 14.9) (Fig

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Collectively, these results suggested that APR-017 and LCS3 treatment induce vulnerability of VRK2-deficient PC cells by GSH inhibition and consequent disruption of the balance between ROS generation and antioxidation