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Interestingly, GLP-1 is also known to decrease the rewarding effects of food intake by interacting with parts of the mesolimbic dopamine system such as the ventral tegmental area (VTA), nucleus accumbens (NAc) [34], and also GABAergic circuits in the laterodorsal tegmental nucleus (LDTg) [35], which makes brain GLP-1R signaling a promising target for the treatment of alcohol and cocaine abuse disorders [36, 37]
2023, McIntyre, Roger S
and 10% in the rest of the world are likely to be treated with GLP-1 therapies by 2035, up from approximately 6% in the U.S
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Indeed, acute activation of GIP signaling in hypothalamic cells results in a decreased food intake in rodents [40]
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