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doi: 10.3390/ijms21103637 219 YuYHeJHHuLLJiangLLFangLYaoGDet al

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The non-enzymatic mitochondrial protein hyperacetylation induced by the increased availability of mitochondrial acetyl-CoA avoids the risk associated with mitochondria overfeeding, restraining the activity of the respiratory complex I and modulating CACT activity

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