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Results GHK Alleviated Neurological Deficits and Promoted Neuron Survival Following ICH To investigate the potential role of GHK post-ICH, we isolated the rat brain 3 days after ICH

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Senescent cells are old, damaged cells that stop dividing but refuse to die, and they can leak inflammatory signals into nearby tissue

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This work suggests that HGF-MET may also protect from parthanatos, a form of caspase-independent regulated cell death occurring especially in neurons and driven by PARP1 hyperactivation (Galluzzi et al., 2018)