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Use of hPSC-derived 3D organoids and mouse genetics to define the roles of YAP in the development of the esophagus

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Proper reconstitution matters equally

Several studies have highlighted the significant benefits of GLP-1 analogs in managing EDs and obesity across multiple domains [16, 17]

During the pathogenesis of OA, JUN directly binds to the promoter of NCOA4 and initializes its transcription, increasing ferritinophagy degradation, leading to ferroptosis and extracellular matrix degradation in chondrocytes