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Given the known off-target effects of APR-246, we investigated whether the mutant-specific effects observed in OVCAR5 cells were attributable to intrinsic differences in redox homeostasis among the parental p53-null cell lines

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245 PI3K is a kinase that can be activated by a variety of external signals to catalyze the conversion of phosphatidylinositol diphosphate to phosphatidylinositol triphosphate

2014), the FAK-paxillin pathway (Chang et al