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Thus, it needs to be cleared out quickly to prevent cell and DNA damage, which can lead to cancerous mutations.[ref] Two well-studied genetic variants have different ways of reaching the same endpoint: ~ an alcohol dehydrogenase (ADH1B) variant that speeds up the conversion to acetaldehyde, thus creating a buildup of acetaldehyde ~ an acetaldehyde dehydrogenase (ALDH2) variant that slows down the conversion from acetaldehyde to acetic acid, again creating a buildup of acetaldehyde

doi: 10.3389/fimmu.2018.00229 86 MeyerDC.A.VRBitarelloBDC.B.DYNunesK

Additionally, PRDX6 acts as a selenium-binding protein that promotes efficient intracellular selenium utilization, thereby sustaining GPX4 expression and activity and indirectly enhancing resistance to ferroptosis (Li et al., 2024b)

Taher, A

In diseases like AD and PD, mitochondrial dysfunction leads to a decrease in the production of ATP while increasing the levels of reactive oxygen species (ROS)

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