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It should be emphasized that this effect does not necessarily mean that Asp itself is a pro-inflammatory factor, but more likely reflects its position in the coupling of energy metabolism and metabolic signaling: when shuttle activity and anabolic demand increase, Asp supply may become one of the limiting factors supporting pro-inflammatory transcription and effector molecule synthesis

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Insulin-like growth factor-I: vitronectin complex-induced changes in gene expression effect breast cell survival and migration