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This interaction was mapped at the hydrophobic domain of G0S2 and the patatin-like domain of PNPLA2, whereby overexpression of G0S2 diminished adipocyte lipolysis, and deletion increased both basal and stimulated lipolysis (52)

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they used self-assembling Helicobacter pylori ferritin nanoparticles with 24 copies of RBD attached via sortase A, named RBDscNP [131] (Table 2)

Solving the -conotoxin folding problem: efficient selenium-directed on-resin generation of more potent and stable nicotinic acetylcholine receptor antagonists