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and group 8: mice administered Dihexa [2]

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Experimental studies involving diabetic ulcer models have suggested accelerated wound closure and improved tissue regeneration following GHK-Cu administration, supporting continued investigation into connective tissue repair and extracellular matrix restoration

849 In animal models of intestinal I/R, ferroptosis occurs primarily during the reperfusion phase due to the inactivation of the GSH/GPX4 pathway

GPX3 is an extracellular enzyme that is particularly abundant in blood plasma, where it contributes to systemic antioxidant protection