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Changes in plasma haptoglobin (Hp) levels are difficult to assess because they are affected by multiple factors: decreased response to needed blood transfusions, potentially functional differences among Hp proteoforms, and the fact that Hp is an acute-phase reactant in humans (Naryzhny and Legina, 2021)

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This obviously requires a mechanism by which the ammonium ion can be transported out of the neurons and into the surrounding astrocytes