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Bieging KT, Fish K, Bondada S, Longnecker R

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Rosenstock, J., Nino, A., Soffer, J., Erskine, L., Acusta, A., Dole, J., Carr, M

Browman RE, Wolf RC

Jacovina and colleagues reported a unique in vivo mechanism suggesting that an annexin A2-deficient mouse rendered hyperhomocysteinemic by dietary means demonstrated impaired fibrinolysis, perivascular fibrin persistence, and attenuated angiogenesis (angiostasis) (4)

J Pediatr Pharmacol Ther 19