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The complex interplay of these factors not only undermines the efficacy of conventional therapies such as ESAs and iron supplementation but also contributes to cardiovascular complications and poor clinical outcomes

Indeed, for ILC2s we observed that this concentration did not precipitate changes in cellular viability ( Figures 3F, G ), and was additionally required to produce maximal suppression of ILC2 activity ( Figures 3H, I )

The comparison is not just academic

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Neuro-Protective role of Metformin in patients with acute stroke and type 2 diabetes mellitus via AMPK/Mammalian target of Rapamycin (mTOR) signaling pathway and oxidative stress