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Thus, despite the limitations of rat studies, the results showed that treatment with BPC 157 led to the recovery of tail function and the resolution of spasticity and improved the neurologic recovery

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This paper reviews the most relevant experimental studies that have contributed to a better understanding of the molecular mechanisms and intracellular pathways involved in cardioprotection induced by GLP-1 and analyzes in depth its potential role as a therapeutic target both in the ischemic and reperfused myocardium and in other conditions that are associated with myocardial remodeling and heart failure