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This article discusses the role of the gut microbiome and redox balance in ASD and notes that a high baseline oxidative burden may make some patients poor responders to glutathione supplementation

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Several kinases can phosphorylate p53 at these sites, including ataxia-telangiectasia mutated (ATM) kinase, ATM- and Rad3-related (ATR) kinase, and checkpoint kinase 1/2(CHK1/2) (Appella and Anderson, 2001)

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ATF6 induction activates ER chaperone protein release such as PDI (Fung et al., 2014), whereas NTZ plays a vital part in the maintenance of protective UPR response via PDI suppression (Di Santo and Ehrisman, 2014