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Further, in the context of heteromeric receptors formed between GluK1-GluK3 receptors, our findings remain valid as both N-glycosylation sites and potential interacting residues are conserved (Supplementary Fig

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ACh regulates Ca 2+ levels and transcription factor expression through muscarinic receptors, while acetylcholinesterase (AChE) promotes the breakdown of ACh

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This not only maximizes the physiological advantages, boosting insulin sensitivity, decreasing inflammation, and increasing fat metabolism, but also empowers the patients to participate in their treatment actively