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Parallel to these endocrine perturbations, IR drives lipotoxicity and systemic inflammation, characterized by increased TNF-, IL-6, and C-reactive protein, which further suppresses insulin sensitivity and antioxidant enzyme expression (78)

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In biochemical and cellular research systems, Sermorelin interacts with the growth hormonereleasing hormone receptor (GHRH-R) expressed on pituitary somatotroph cells, initiating intracellular signalling cascades that regulate growth hormone synthesis and release

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FUS/TLS assembles into stress granules and is a prosurvival factor during hyperosmolar stress