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BPC-157 is believed to interact with the nitric oxide (NO) system, angiogenic growth factors, and cellular signaling pathways like FAK-paxillin and VEGF, which are involved in cell migration, cytoskeletal reorganization, and vascular modulation

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(2010) demonstrated through knockout experiments that Nrf2-deficient mice exhibited heightened susceptibility to the MPTP-induced PD model, resulting in significantly lower survival rates of dopaminergic neurons