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By coupling agonism at the GLP-1 receptor with simultaneous agonism at the GIP receptor, a related incretin whose physiological role had been the subject of decades of competing theories, Eli Lilly produced a molecule whose efficacy in trial conditions exceeded semaglutide by a margin too large to dismiss as noise or trial-design artifact

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Since HCV isolates apart from JFH1 require massive adaptation to allow infectious particle production 22 , including the GLT1 isolate 21 , we employed chimeric genomes encoding the JFH1 replicase fused to the pre- and post-transplant structural proteins to assess changes in infectious particle production 23 (Fig

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