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Glutathione is a small peptide molecule, composed of three amino acids, cysteine, glutamic acid, and glycine

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We showed that our model accurately reproduces published clinical and experimental data on the dose-dependent time course of acetaminophen in the plasma, the accumulation of acetaminophen and its metabolites in the urine, and the depletion of glutathione caused by conjugation with the toxic product

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These results suggest that SV enhances antioxidant capacity by modulating the KEAP1/NRF2/HO-1 pathway