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Experimental evidence is ongoing to explore and unravel all these possible beneficial mechanisms, while the combination of both drug categories (at least in patients with T2D) with or without other therapeutic strategies seems promising

GLP-1 receptor agonists like semaglutide work by slowing gastric emptying and increasing satiety signals in the brain, which fundamentally changes how your body responds to sugary and high-calorie foods

Before, I would be tempted [to overindulge in food], but now there's just no temptation there for me

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The GI side effects that plague so many semaglutide users, the nausea, the bloating , the constipation, the diarrhea, are not random bad luck