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However, it also interacts with cardiac-specific receptors, including CD36, which may mediate its cardiovascular effects independently of GH

Reactivation of the p53 tumor suppressor pathway by a stapled p53 peptide

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It should only be used for in vitro laboratory research

Accordingly, the two-domain ligand binding model suggests that the N-terminal part of the peptide hormones engage the TM and ECL region of the receptor leading to activation and signal transduction 13