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In contrast, if RBCs were loaded with human hexokinase, they showed a doubled glycolytic activity, a normal hexose monophosphate pathway, and normal ATP and 2,3-BPG concentrations (Magnani et al., 1988)

The production of ROS is elevated in tumor cells as a consequence of increased metabolic rate, gene mutation and relative hypoxia, and excess ROS are quenched by increased antioxidant enzymatic and nonenzymatic pathways in the same cells

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