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GSH regulates mTORC1 signaling and the ISR during cystine starvation Given that cystine and GSH balance are tightly coupled, we next evaluated whether GSH is a modulator of cystine-induced cell signaling

Our results provide further evidence of a D2R/D3R-dependent inhibitory mechanism that regulates -cell hormone secretion, and that disruption of this signaling by APDs increases glucagon release

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In healthy and diabetic subjects, the co-agonist displayed to improve glucose tolerance and insulin secretion, although no data regarding bodyweight, lipid metabolism, or liver function were reported (78)

Most likely reality: microdoses produce small metabolic improvements - subtle visceral fat changes, modest improvements in insulin sensitivity, minor reductions in inflammatory signaling - that compound over time into meaningful clinical benefits