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Cellular antioxidant defence: Direct protection of cells and DNA from oxidative stress damage inside the cell itself, not just in the bloodstream

For instance, MCT1 inhibition in glycolytic tumors induces a metabolic catastrophe that triggers caspase-dependent apoptosis alongside GSH depletion-mediated ferroptosis, thereby yielding enhanced antitumor efficacy relative to the activation of either pathway alone (142)

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Once desired results are achieved, maintenance sessions may be recommended every few weeks or months