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However, clinical translation of this mechanism remains in its infancy

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In the same research, it also reduced markers of apoptosis (programmed cell death) in those cells, keeping them alive and functional longer

By analogy with findings in other cell types, MAMs may potentiate the activity of Ca 2+ -dependent enzymes in the TCA cycle by regulating Ca 2+ transfer between the endoplasmic reticulum and mitochondria, thereby potentially enhancing mitochondrial OXPHOS efficiency and ATP production.However, direct evidence supporting this mechanism in ovarian cells is currently lacking

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