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Blood pressure is tightly regulated through the renin-angiotensin-aldosterone system (called RAAS)

Although biochemically separated from the GSH system, the Trx system shares NADPH as a common reducing cofactor and serves as an alternative pathway for reducing disulfide bonds, making this system especially relevant if the GSH system has been compromised or rendered dysfunctional ( Under oxidative stress these three systems work in concert to detoxify ROS and lipid peroxides, maintain protein structure and function via thiol repair, prevent activation of redox-sensitive inflammatory pathways, and preserve mitochondrial membrane integrity and prevent apoptosis

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The first generation of these therapies focused on GLP-1 receptor agonism alone

Patient retention rates average 78% at six months, with most discontinuations due to cost rather than efficacy concerns

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