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Here, we aim to enlist computations to discover how same pathway and same-gene mutations, and overexpression can preferentially lead to cancer and NDDs phenotypes, with the overarching goal of applying the learned knowledge to identify likely culpable genes/mutations (Figure 1B)

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albicans and forces it to change its metabolic mode, thereby weakening its toxicity) and as a direct secretion of molecules with antifungal activities (e.g., lactic acid, short-chain fatty acids, hydrogen peroxide, bacteriocin-like substances, and biosurfactants) [25, 159, 162, 163]

Four peptide hormones specific decrease (up to 97%) of human prostate carcinoma cells

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